The rate-limiting step of the protamine-induced adenosine triphosphatase activity of adenosine triphosphate-G-actin

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The rate-limiting step of the protamine-induced adenosine triphosphatase activity of adenosine triphosphate-G-actin.

The release of Pi from the Pi-G-actin-ADP complex is the rate-limiting step in the ATPase activity that is shown by ATP-G-actin in the presence of protamine.

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The magnesium ion-dependent adenosine triphosphatase of myosin. Two-step processes of adenosine triphosphate association and adenosine diphosphate dissociation.

The kinetics of protein-fluorescence change when rabbit skeletal myosin subfragment 1 is mixed with ATP or adenosine 5'-(3-thiotriphosphate) in the presence of Mg(2+) are incompatible with a simple bimolecular association process. A substrate-induced conformation change with DeltaG(0)<-24kJ.mol(-1) (i.e. DeltaG(0) could be more negative) at pH8 and 21 degrees C is proposed as the additional ste...

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Adenosine Triphosphate Cleavage during the G-actin to F-actin Transformation and the Binding of Adenosine Diphosphate to F-actin.

Since the discovery by Straub and Feuer (1) and by Laki, Bowen, and Clark (2) that adenosine triphosphate bound to Gactin is transformed to adenosine diphosphate and inorganic phosphate during polymerization of actin, it has become increasingly clear that the chemical changes in the nucleotide are related to the change in the physical state of the protein. Barany, 13iro, Molnar, and Straub have...

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Studies on Myofibrillar Adenosine Triphosphatase with Calcium-free Adenosine Triphosphate.

It has been reported by several groups of workers that incubation of the relaxing factor granar of muscle with magnesium and adenosine triphosphate results in the formation of a soluble relaxing substance capable of inhibiting the magnesium-activated adenosine triphosphatase activity of myofibrils or actomyosin (3-6) and the contraction of single glycerinated muscle fibers (7). In addition, Uri...

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Inhibition of Adenosine Triphosphatase Activity

Triphenylsulphonium ions inhibit mitochondrial oxidative phosphorylation and adenosine triphosphatase activity. The site of action is on the soluble F1 adenosine triphosphatase component. Triphenylsulphonium ions also inhibit electron transfer in the NAD-cytochrome b region of the respiratory chain. In both types of inhibition, triphenylsulphonium ions are effective at low concentrations, half-...

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ژورنال

عنوان ژورنال: Biochemical Journal

سال: 1979

ISSN: 0264-6021

DOI: 10.1042/bj1830475